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This effect could explain instances of constrictive arachnoiditis and sterile meningitis reported after repeated intrathecal TRIAM injection four However, we should point out that CSF investigations relating to markers for cell injury, such as neuron-specific enolase, S-one hundred, neurofilament heavy chain, tau protein supplied no convincing proof of neuronal or SC harm after the intrathecal TRIAM therapy 35 The interplay of drugs utilized intrathecally with the MHCII molecule and its function for sterile meningitis remains to be further investigated as other substances with non-crystalline structure may also result in this facet effect 36 As we did not investigate the effects of TRIAM in neuron-SC cocultures relating to myelination, we are able to only postulate that the late impact on myelination proven on day 23 p.i. could be attributed on an improved SC myelination operate.
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